Managing acute gout episodes

The main causes are: - excessive consumption of foods rich in uric acid - consumption of diuretic drugs - hereditary factors (genetic inheritance) - falling medicines - obesity - renal failure - vitamin B3 . The gout crisis is initially manifested by sudden and localized pain at the level of the toe (the toe), very intense, permanent, usually occurring during the night, accentuated by the slightest touch and any attempt to mobilize. The joint is swollen, red, hot and painful. The most affected areas are: toes, knees, ankles, hands and elbows. If the disease becomes chronic and is not treated in time, uric acid begins to deposit as urate crystals in various organs, and the most commonly affected organ is the kidney, where the so-called \. Investigations to be done are not costly or put the patient's life at risk.

It starts by doing a full blood test, analyzes that include determining uric acid in the blood, then if it has an elevated value, a summary urinalysis is also performed to see if there is (urine sedimentation and sediment analysis . If the symptoms persist and uric acid keeps growing, synovial fluid (inside a joint) will be sampled using the ultrasonically guided puncture and to see if microscopic crystals of sodium urate. Gout crises can relapse on a regular basis but are largely influenced by food overdoses and alcohol consumption. The main complication is the formation of urinary calculi and the onset of painful kidney colic and the symptoms described above. In the absence of treatment, or in the case of incorrect treatment, goutosis occurs, which are described as white uric acid deposits under the skin, which are sensible to palpation and are encountered in the small joints of the fingers, large joints, .

The most severe complication is renal impairment, since it can gradually lead to renal failure if it is not treated in time and if the uric acid value in the blood is not balanced. Gout crisis is treated with a drug that is given only during painful pushing. Sometimes this drug is also considered a test to confirm the diagnosis in the sense that the symptomatology is or not. Then continue with the anti-inflammatory, which helps to reduce inflammation and reduce symptoms that are hard to bear, and to lower blood uric acid levels and maintain a target value, medications called hypersulin are administered to accelerate the elimination of uric acid via the . It is also worth noting that patients may experience a new gout attack at the start of treatment that decreases ugly because the drug stimulates detachment by partial dissolution, although it is said that increasing the dose decreases the risk of new gout.

Treatment should be maintained over a long period of time to reduce the risk of recurrence and once the target and steady-state concentration of uric acid is reached, the analyzes should be repeated every 6 to 6 months to determine whether the therapeutic target is maintained or . From the moment the patient is therapeutically balanced, control can be done annually or even two years if there are no new gout crises. Prevention of gout access depends on each individual patient and its consistency with regard to the regimen to be followed. Dietary measures imposed are not the most drastic, they sell to reduce the consumption of purine-rich foods that result in uric acid, limiting fat consumption and stopping if necessary, and hypocaloric regimen in case of overweight. Water consumption is indicated to dilute urine and prevent the formation of kidney stones by removing uric acid crystals.

Consumption of black cherries and would bring a beneficial benefit to patients because the substances found in these fruits and vegetables have anti-inflammatory properties (reduce inflammation). .